Last week we discussed how the physiological determinants of diastole play a role in the evaluation method of determining diastolic function. This week, let’s talk about how pathophysiological factors can alter the findings in the presence of diastolic dysfunction.
As the year comes to an end, we would like to thank all of our readers and clients for another successful year at CardioServ. We started the year off with goals to
The Intersocietal Accreditation Commission updated the standards and guidelines for Adult Echo accreditation in July 2017 with a December 1, 2017 effective date. The main changes to the adult echo standards is the mandate (versus option) to evaluate diastolic function on all studies, and to report on it. With this requirement followed the requirements for all ultrasound equipment to have Tissue Doppler Imaging capabilities.
This year for Ultrasound Awareness Month, we asked you all to recognize yourself or a fellow sonographer or physician by submitting a story about a time you saved a day or made a difference. Here at CardioServ, we believe that each and every sonographer is a hero in their own way and we want to share your stories!
Diastolic function is a complex process that simply refers to how well the heart performs during diastole, as blood is suctioned from the left atrium (LA) towards the apex of left ventricle (LV) at a low-pressure filling state. When the heart is unable to fill properly, filling pressures increase in order to allow proper filling and keep required cardiac output state. When these filling pressures increase, diastolic dysfunction occurs.
Over the past weeks, we have gone back to the basics to have a good understanding of what occurs during diastole in order to properly evaluate diastolic function. If you’ve missed our prior blogs, you can find them here:
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